Vaccines: Immune cells transport aluminum adjuvant to sites of injury

This study shows that aluminum-engorged macrophages can survive long enough to transport their cargo to sites of injury and inflammation (including the brain), creating chronic inflammation and setting off a feedback loop fed by further inoculations.    Unfortunately aluminum isn’t the only worry.


Within clinical vaccinations, recombinant antigens are routinely entrapped inside or adsorbed onto the surface of aluminium salts in order to increase their immunological potency in vivo. The efficacy of these immunisations is highly dependent upon the recognition and uptake of these complexes by professional phagocytes and their subsequent delivery to the draining lymph nodes for further immunological processing. While monocytes have been shown to internalise aluminium adjuvants and their adsorbates, the role of macrophages in this respect has not been fully established. Furthermore, this study explored the interaction of THP-1 macrophages with aluminium-based adjuvants (ABAs) and how this relationship influenced the survival of such cells in vitro. THP-1 macrophages were exposed to low concentrations of ABAs (1.7 μg/mL Al) for a maximum of seven days. ABA uptake was determined using lumogallion staining and cell viability by both DAPI (4′,6-diamidino-2-phenylindole) staining and LDH (lactate dehydrogenase) assay. Evidence of ABA particle loading was identified within cells at early junctures following treatment and appeared to be quite prolific (>90% cells positive for Al signal after 24 h). Total sample viability (% LDH release) in treated samples was predominantly similar to untreated cells and low levels of cellular death were consistently observed in populations positive for Al uptake. It can thus be concluded that aluminium salts can persist for some time within the intracellular environment of these cells without adversely affecting their viability. These results imply that macrophages may play a role in the systemic translocation of ABAs once administered in the form of an inoculation….

Birth: immediate cord clamping can cause brain damage, either hemorrhage or asphyxia:

Aluminum from HepB vaccine (routinely given at birth to babies of healthy mothers for NO DEFENSIBLE REASON WHATSOEVER, there goes your “evidence base”) is transported to such damaged sites.

This is also Forrest Maready’s thesis:

Recent scientific research has shed light onto what happens when metals like aluminum or mercury are put inside your body. Your body’s white blood cells surround it, because they know metals are dangerous. With a virus or bacteria, the white blood cells could easily destroy it. But it’s metal, so the white blood cell can’t do anything with it. They surround it, but nothing happens. It doesn’t get excreted through your kidneys over time. You don’t pee it. You don’t poop it. Rabbits showed us this. Injected metal just accumulates in your body, usually within muscle tissue.

If we go back to those disorders, Bell’s Palsy, Crohn’s, Autism, Hashimoto’s – they all seem very different, but their onset often follows the same pattern. In fact, if you look at any of the auto-immune disorders, and even many neurological disorders like Parkinson’s or ALS, they all seem to start after a significant immune activation event. Something that really fires up your immune system: an infection, surgery, stress, physical exertion or pregnancy.

When your body’s immune system becomes activated, there’s one very important thing it does. It signals for help. From what? From white blood cells. These white blood cells answer the call for help, but unfortunately, because we now inject metal into our bodies, many of the white blood cells bring a deadly payload with them, like ALUMINUM. And when aluminum’s delivered to a part of your body that needs help, very bad things happen. Which causes a signal for more help. Which brings more aluminum. And on and on it goes….