Takeaway: vaccines more or less work against the targeted organism but diminish immunity for non-targeted organisms http://thoughtcrimeradio.net/2012/01/flu-shot-may-weaken-immune-system/ , probably for the same reason that vaccine immunity fades over time and often results in autoimmune diseases: they’re throwing a monkey wrench into a finely tuned internally-coherent system which has co-evolved with its environment for millennia. Vaccine viral contaminants are probably also involved, as well as the likely correlation between adhering to standard vaccination guidance and following other standard health regimens such as sunshine avoidance, lack of breast feeding and toxic food.
Here’s the links from today’s show!
You can’t touch this #HWHero;
Shocking new info about #Polio in the Philippines;
Deconstructing the Dogma on #PetHealth, with Dr. Dym
Rockland County Healthiest:
It is exceedingly rare for the public health community to admit to any problems with vaccination. Every so often, however, circumstances force officials into making just such an acknowledgement. The current debacle seeping out into the news–which is actually a long-running tale minted anew–is that oral polio vaccines are “spawning virulent strains” of polioviruses. The alarming surge in vaccine-derived polio cases presents vaccine planners with a “quandary” or “conundrum”–because “The very tool you are using for [polio] eradication is causing the problem.”
The oral polio vaccine (OPV) is in use around the world and constitutes the “workhorse” of global polio eradication efforts due to its low cost and ease of administration. The OPV contains live but weakened polioviruses that match up to wild polioviruses. Vaccine researchers have long known that these OPV-derived viruses can themselves cause polio, particularly when they get “loose in the environment.” In settings with poor sanitation and iffy hygiene, the vaccine viruses can easily “find their way into water sources, and onto contaminated hands or foods,” where they can then launch a self-perpetuating chain of transmission. Researchers concede that an OPV virus “can very rapidly regain its strength if it starts spreading on its own,” acquiring “mutations that make it basically indistinguishable from the wild-type virus.” In other words, there is no meaningful difference between a wild and OPV-derived poliovirus “in terms of virulence and in terms of how the virus spreads.”
Until recently, the OPV was trivalent, containing three vaccine serotypes corresponding to the three types of wild polioviruses (types 1, 2 and 3). In 2015, however, global public health agencies declared wild poliovirus type 2 eradicated and, in 2016, they decided to oversee a closely coordinated 155-country “switch” to a bivalent oral vaccine that includes only types 1 and 3. The ostensible rationale for this global maneuver was to take vaccine-derived type 2 virus out of the running; if wild type 2 viruses had disappeared, “the thinking went,” it would be “unethical to expose children to the risk the vaccine viruses posed,” particularly since type 2 had been responsible for close to 90% of the vaccine-derived polioviruses circulating since the year 2000. However, in a what-could-possibly-go-wrong scenario, the lingering background presence of the type 2 vaccine virus has prompted a spectacular backfiring of the “switch,” with vaccine-derived polio outbreaks emerging in numerous countries.
The Global Polio Eradication Initiative (GPEI) is a combined effort of the World Health Organization (WHO), UNICEF, the U.S. Centers for Disease Control and Prevention (CDC), the Bill & Melinda Gates Foundation and Rotary International. In pre-“switch” computer modeling, the GPEI’s scientific advisors predicted that some vaccine-virus-derived outbreaks would inevitably occur. However, they confidently asserted that they would be able to “quickly squelch” any such outbreaks through–what else?–the “judicious use of a new live vaccine…effective against only type 2.” In effect, they advised taking a “gamble” that the monovalent vaccine “would not spawn new outbreaks of its own.”
In Africa, at least, the gamble has failed. The frequency with which type 2 vaccine-derived outbreaks are occurring has far exceeded projections–and the rush to administer the new monovalent type 2 vaccine appears to be exacerbating rather than stemming the problem. In an astonishing admission, a CDC virologist has stated that due to the stop-gap use of the new type-2-only vaccine, “We have now created more new emergences of the virus than we have stopped.” Another vaccine expert has remarked, “if you just keep trickling in with a little bit of [monovalent] vaccine every time you think you have a problem all you’re doing is reseeding [more transmission chains].”
There had been no cases of wild poliovirus on the African continent since September 2016, but by July 2019, the WHO was cautioning that there was a high risk of ongoing type 2 vaccine virus spreading across Africa. Outbreak investigators have been documenting an uptick in circulating vaccine-derived poliovirus type 2 in both human and environmental samples since mid-2017 (two years after the “switch”), generally obtaining human samples either from children presenting with acute flaccid paralysis (AFP) or from “healthy community contacts.” Although the WHO describes polio as just one of AFP’s possible causes, African labs have been isolating type 2 vaccine virus in case after case of AFP.
To date, surveillance reports have noted the presence of the vaccine-derived type 2 poliovirus in Angola, Cameroon, Central African Republic, the Democratic Republic of the Congo, Ethiopia, Ghana, Kenya, Mozambique, Niger, Nigeria, and Somalia. In Nigeria, type 2 has spread from the north of the country to Lagos–Nigeria’s largest and most densely populated city. In Ghana, soon after investigators found type 2 vaccine viruses in sewage in the capital of Accra, a toddler 400 miles away was diagnosed with vaccine virus paralysis–representing Ghana’s “first ever” reported outbreak of type 2 vaccine-derived poliovirus.
On September 19, ABC News announced a polio outbreak in the Philippines, caused–as confirmed by WHO and UNICEF–by “vaccine-derived poliovirus type 2.” The “outbreak”–which has thus far affected just two children and some sewage samples–is unfolding two decades after the WHO declared the Philippines to be polio-free. Blaming the situation on the decline in public acceptance of routine vaccination–following the rollout of a dangerous dengue vaccine that killed as many as 119 children in 2016-2017–the two global health agencies have sternly asserted that the only way to get back on track with polio is to vaccinate at least 95% of children under age 5. The Philippine government is complying, launching an aggressive polio vaccination campaign ….
The graph is from the Ratner report (1), the transcript of a 1960 panel sponsored by the Illinois Medical Society, on which sat three PhD statisticians and an MD, met to discuss the problems with the ongoing polio vaccination campaign.
The polio vaccine was licensed in the U.S. in 1954. From ‘50 thru ‘55, the striped and clear portions of the bars represent about 85% of the reported cases, or 30,000 per year, on average. Those cases were automatically eliminated by two radical changes the CDC made to the diagnostic parameters and labeling protocol of the disease as soon as the vaccine was licensed – 30,000 cases a year we were subsequently told were eliminated by the vaccine.
That success, held aloft as a banner of the industry, is an illusion. The CDC has an awesome power of control over public perception, sculpting it from behind closed doors in Atlanta, with the point of a pen.
Over the last sixty years in the U.S., more than a million cases of what would have been diagnosed as polio pre-vaccine – same symptoms – were given different labels.
The change didn’t stop there, however. As addressed in the Ratner report, they also changed the definition of a polio epidemic, greatly reducing the likelihood that any subsequent outbreaks would be so labeled – as though the severity, or noteworthiness, of paralytic polio had halved, overnight. It’s summed up thusly in the report:
Presently , a community is considered to have an epidemic when it has 35 cases of polio per year per 100,000 population. Prior to the introduction of the Salk vaccine the National Foundation defined an epidemic as 20 or more cases of polio per year per 100,000 population. On this basis there were many epidemics throughout the United States yearly. The present higher rate has resulted in not a real, but a semantic elimination of epidemics.
And that’s precisely what happened to polio: not a real, but a semantic elimination of the disease.
In the decades following the release of the vaccine, additional changes were made to the diagnostic parameters of the disease, changes involving analysis of cerebrospinal fluid and stool and additional testing (2) , each succeeding change making it less and less likely that a diagnosis of paralytic polio would result.
And, critically, before the vaccine was licensed polio diagnoses were made clinically and accepted from around the nation, duly reported to the American public annually as polio, no lab analysis required, while after it was licensed only the CDC was – and is – allowed to issue confirmations of paralytic polio – all suspected cases had to be sent to them for analysis and testing. (3)
Again, perception is key. Because of the persistent pre-vaccine news coverage of the disease, including film footage of paralytic polio victims in leg braces, or immobilized, strapped to huge, inclined boards, or housed in foreboding iron lungs, the public pictured the thousands of kids reported with polio each year as suffering terribly, when in truth the pictures involved only a fraction of a percent of the diagnosed cases.
Moreover, while for many the perception was that the iron lung was a permanent fixture, in the majority of cases the machine was needed only temporarily – generally about one to two weeks. (4)
The arbitrariness of the change in the diagnostic parameter of paralytic polio, from one day of paralysis to two months, resulting specifically in the elimination of all the cases represented by the striped portions of the bars in the graph, is remarkable. Indeed, the very idea that the length of time you’re ill determines the disease is remarkable!, and flies in the face of the science of virology.
Were you to apply the same logic to measles diagnostics, for instance, and add the requirement of a rash that lasts ten days, the disease would be eradicated, since the measles rash lasts from three to five days. To the point, had they made the requirement three months of paralysis instead of two, several additional thousands of cases of paralytic polio would simply and immediately have fallen off the diagnostic plate, hastening the illusion of complete eradication.
All of the non-paralytic cases, represented by the clear portions of the bars in the graph, and which pre-vaccine were the majority of cases reported simply as polio each year, were discarded completely!, reclassified. A search through public health department disease statistics reveals that in the U.S. those cases were basically handled as they were in Canada:
It may be noted that the Dominion Council of Health at its 74th meeting in October 1958 recommended that for the purposes of national reporting and statistics the term non-paralytic poliomyelitis be replaced by ‘meningitis, viral or aseptic’ with the specific viruses shown where known. (5) …